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FIGURE 3 Together, these findings suggest that HA + GSH-C4 induces a transient, reversible cytokine activation with repair-associated Th1/Th17 features, with IFN- and IL-12p70 persisting even after crossover to HA alone

Increased levels of oxidative stress markers may suggest ongoing tissue damage and inflammation, assisting in determining the timing and intensity of antioxidant therapy as part of metabolic interventions (139)

Missing data on key clinical factors (such as body mass index and type 2 diabetes duration) may contribute to residual confounding, leaving uncertainty about whether the observed association is causal

Body weight influences needslarger individuals generally require more fluid, though personalized targets should be developed with healthcare providers

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Mitochondrial dysfunction: A potential therapeutic target to treat Alzheimer's disease