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Preventive treatment of astaxanthin provides neuroprotection through suppression of reactive oxygen species and activation of antioxidant defense pathway after stroke in rats

Researchers cite more than four decades worth of research into GHK-Cu, including non-human and human studies that show that the peptide is safe and non-toxic [1]

The accumulation of ROS and RNS results in lipid peroxidation, where polyunsaturated fatty acids (PUFAs) in neuronal membranes undergo oxidative damage, compromising cell integrity and function

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