This helps to minimize the counter-regulatory action of GH against higher daytime insulin levels (and its potential for increasing insulin resistance)
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In summary, we found that high glucose increases ROS production in PSCs, which in turn activates PSCs by upregulating the TGF signalling pathway to promote their proliferation and migration
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In addition, we should also consider the relationship between c-Met inhibitors and liver disease, although there is no clinical evidence that c-Met inhibitors worsen liver function
