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In placental development, HO-1 is highly expressed and fulfills essential physiological roles: it is enriched in trophoblast cells, where it modulates EVTs invasion via upregulation of peroxisome proliferator-activated receptor (PPAR) [30, 70], a key regulator of trophoblast differentiation and spiral artery remodeling

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However, the modern world puts a heavy load on our internal systems