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In people with prediabetes, this mechanism can prevent or delay the onset of type 2 diabetes

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It is also the appropriate choice for study designs where researchers want to isolate the contribution of GIPR agonism to metabolic outcomes with semaglutide as the GLP-1R mono-agonist control and tirzepatide as the dual-agonist comparator

Whole brain microscopy following the injection of fluorescently labeled semaglutide revealed that semaglutide is present in parts of the hypothalamus, including the DMH, ARC, PVH, medial mamillary nucleus, and SON [59]

Rigault, C

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