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Studies indicate that despite its acetyl group structure designed to improve bioavailability, ALCAR metabolism still produces trimethylamine N-oxide (TMAO) as a metabolic byproduct, though this occurs at lower levels than with standard carnitine forms [5]
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Current treatment includes a low fat, high carbohydrate diet and avoidance of fasting which dramatically improves prognosis allowing long term survival
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Muscle and heart disease: including muscle weakness, cardiomyopathy (myocardial hypertrophy or dilation), and arrhythmias.Nervous system effects: may manifest as brain damage, seizures, intellectual disability, ataxia, etc
26 professional soccer players were divided into two groups and administered either 3g, or 4g of l-carnitine
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