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PARP-1 involves in UVB-induced inflammatory response in keratinocytes and skin injury via regulation of ROS-dependent EGFR transactivation and p38 signaling
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Water, Centella Asiatica Extract (20.5%), Butylene Glycol, Glycerin, Dipropylene Glycol, Niacinamide (20,000ppm), Hydroxyethyl Urea, Sodium Hyaluronate, Allantoin, Panthenol, Glutathione (1,000ppm), Nelumbo Nucifera Extract, Artemisia Annua Extract, Oryza Sativa (Rice) Extract, Saccharomyces Ferment, Solanum Melongena (Eggplant) Fruit Extract, Melaleuca Alternifolia (Tea Tree) Leaf Extract, Hydrolyzed Collagen, Beta-Glucan, Hippophae Rhamnoides Fruit Extract, Adenosine, Madecassic Acid, Asiaticoside, Asiatic Acid, Squalane, Caprylic/Capric Triglyceride, Hydrogenated Lecithin, Glyceryl Stearate, Cholesterol, Xanthan Gum, Propanediol, Hydroxyacetophenone, 1,2-Hexanediol, Ethylhexylglycerin, Dipotassium Glycyrrhizate, Styrene/Acrylates Copolymer, Citric Acid, Sodium Citrate
There is a therapeutic rationale behind this combination
Particularly, the significant variation in inducing conditions such as IL-1, oxidative stress, and iron load across different studies makes it unclear whether SLC7A11 downregulation is a universal mechanism in OA, lacking systematic assessment of consistency across models
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