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Accordingly, Masson staining revealed that recombinant CXCL10 treatment sharply increased fibrosis in the CXCL10 group, whereas IL18R1 antibody treatment reversed fibrosis to levels close to those of the control in the 10+18ab group (Fig

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& Georgiou, G

148 While IL-4R knockout (KO) in hepatocytes indeed reduces the direct response to IL-4, it does not completely inhibit liver regeneration

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235,360,361 Another hypothesis is that hepatocytes undergo a transient epithelialmesenchymal transition to escape the suppressive effects of elevated TGF- levels