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Moreover, dysbiosis increases the production of ROS within cells through various mechanisms, including mitochondrial dysfunction, activation of NADPH oxidases, and inhibition of antioxidant enzymes (57)

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Th17 cells, whose proliferation is closely connected to gut dysbiosis, produce IL-17 A and GM-CSF, which not only activate microglia but also damage the BBB by acting on endothelial IL-17RA, resulting in a self-reinforcing permeability loop

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The MNADK is predominantly expressed in the liver, brown fat, heart, muscle, and kidney in mice [54, 56, 57]