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Except for 16S rRNA sequencing, all in vitro studies were based on at least three repeats and in vivo data on 510 mice per group with at least two independent repeats to achieve significant differences

Importantly, IGF-I and its binding proteins exert acute anabolic effects on metabolism as well as mediating myoblast proliferation, differentiation, and apoptosis[2]

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With regard to mitochondrial energy metabolism, these cells have a higher requirement for OXPHOS via AMPK activation than the T helper 17 (Th17) cell subset (62)