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Activation of GLP-1R by its agonist liraglutide (CM + Lira) markedly suppressed the expression of CGRP compared with the corresponding level in the CM group (Fig

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In severe cases that do not respond to first-line approaches, external therapies that modulate nerve function and more invasive treatments such as medications, deep brain stimulation, and cochlear implants may be considered

This mechanism links Epithalon to cellular longevity pathways, as telomere attrition is a documented biological aging marker

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