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This review highlights oxidative stress as a major contributor to brain aging and neurological disease, with the brain particularly vulnerable due to its high metabolic activity

GLP- 1's anti-apoptotic and proliferative effects are also mediated by the PI3 K/Akt axis through the transactivation of the epidermal growth factor receptor (EGFR), which is connected to GLP- 1 receptor through c-Src activation and the generation of endogenous EGF like ligands [59]

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Brain histamine triggers mast cells and induces inflammation