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SAP triggers the release of a multitude of inflammatory factors, including the caspase-1-dependent liberation of IL-18 and IL-1

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PT is also prolonged in some patients with liver disease because of the presence of dysfibrinogenemia, an abnormal form of fibrinogen that does not clot normally, and that may predispose to thrombosis

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These platelets are then re-injected into targeted spinal areas to support repair and reduce inflammation