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We identify mitochondrial trifunctional protein- (MTP) as a binding partner of GLP-1(2836) and demonstrate that the ability of GLP-1(2836) to shift substrate utilization from oxygen-consuming fatty acid metabolism toward oxygen-sparing glycolysis and glucose oxidation and to increase cAMP levels is dependent on MTP

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Molecular basis for Rac2 regulation of phagocyte NADPH oxidase

Simultaneously, the original creators of our GLP-1s, Novo Nordisk and Eli Lilly, took an offensive strategy against the compounding industry

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