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ATP directly facilitates the stability of membrane cytoskeleton connections by modulating spectrin actin dynamics and preserving the attachment of integral proteins, such as band 3, to the cytoskeleton (28)

These findings demonstrate that SAC inhibits Erastin-induced ferroptosis through activation of the Keap1/Nrf2/GPX4 signaling axis

Creatine phosphate provides the rapidly available ATP buffer that supports the high-intensity exercise most likely to upregulate endogenous MOTS-c

These mechanistic studies emphasize that targeting gut-driven immune responses could modulate neuroinflammation and improve stroke outcomes