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Among these factors, the dynamic balance of iron and the abnormality of lipid peroxidation are the most important to induce ferroptosis, while the functional state of System X c and the activity level of GPX4 are the key pathways to regulate ferroptosis

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Together, these findings reveal that ferroptosis suppression via NRF2 activation or ACSL4 inhibition underpins resistance to standard targeted therapies in ccRCC