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More significantly, MOTS-c administration shifted the trajectory of age-related metabolic decline, indicating that the peptide's benefits are not limited to preventing dysfunction in young animals but can partially reverse established decline [2]

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This cardiomyopathy is characterized by dysfunction of cardiac mitochondria, a decrease in plasma selenium, decreased cardiac GPx1 expression, increased oxidative stress in the myocardium as reflected by an increase in 8-hydroxy-2-deoxy-guanosine content of nuclei, and decreased whole blood GPx1 activity [24, 25]

The reviewers concluded there was insufficient evidence to recommend silymarin for hepatitis B and C (Rimbaldi, Cochrane Database Syst Rev, 2007) [29]

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