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More stable than endogenous GLP-1 due to its albumin-binding abilities, semaglutide has an extended duration of action and is thus suitable for once-weekly administration [4, 5]

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This review of the evidence indicating whether UA may have a protective role in the development and/or progression of PD draws on the findings of reduced serum UA in PD, critically assesses the more equivocal genetic results, and discusses experimental observations that UA may provide protection against the pathogenic mechanisms of PD

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A biomarker that shifts in the expected direction feels like progress, and it can justify advancing a program, but it is evidence of activity, not of efficacy