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3.3 Subchondral bone remodeling and ferroptosis Subchondral bone remodeling is a hallmark pathological feature in advanced stages of OA, typically characterized by bone sclerosis and osteophyte formation

It also acts as an antioxidant and scavenges free radicals

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revealed that in an ISO-mediated model of myocardial infarction, direct overexpression of the Nrf2 gene enhanced GSH levels in cardiomyocytes and guarded cardiomyocytes against oxidative damage and lessened the occurrence of ferroptosis [202]