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The mechanism of semaglutide can be elaborated under the following actions: By binding to GLP-1 receptors on beta cells in the pancreas, semaglutide stimulates insulin release in response to high glucose levels

Chronic low-grade inflammation drives pain, easy bruising, and progressive fibrosis

It also helps control kidney urate transporters - it increases ABCG2 (which helps remove uric acid) while decreasing URAT1 and GLUT9 (which keep uric acid in the body) [9]

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