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Given the potential of reduced gliotoxin to thiolate cellular proteins, we speculate that reduced gliotoxin may be sequestered into intracellular vesicles where it is converted to the oxidized form, by an unidentified activity, prior to release from the cell by an exocytotic mechanism complementary to GliA-mediated efflux (Figure 7)

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While some laboratory animal studies have been cited in support of purported TB-500 benefits such as accelerated healing, these findings have yet to be examined in clinical trials