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To summarize, mitophagy is crucial in the management of DKD, and an imbalance of mitophagy can lead to an increase in apoptosis, cellular iron death, accelerated cellular senescence, ROS production, as has the intensity of inflammatory responses, activation of the NLRP3 inflammatory vesicles, an augmentation in oxidative stress, and a decrease in mitochondrial biogenesis, but these can be solved through drugs or compounds such as TSF, JCYSTL, and metformin (Figs
Overall, it can be theorized that alleviation of behavioral deficits was induced by a broad oxidative neuroprotective effect rather than modulation of neuronal activity or a single neurotransmitter pathway
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