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In comparison to the wildtype and vtc2-1 mutant, the pad2-1 mutant seems to compensate its glutathione deficiency by maintaining comparatively high levels of Asc in peroxisomes and by forming an additional layer of palisade cells (Heyneke et al
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(2021) 131:e143137
Once broadly recommended for prevention, todays guidelines only suggest it for specific higher-risk people
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Protein mediated oxidative stress in patients with diabetes and its associated neuropathy: correlation with protein carbonylation and disease activity markers
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