In the perspective of protective mechanisms of aging cardiomyocytes, inhibition of BMAL1/Clock by NAD+ reduced NF-B binding to DNA, which further decreased the formation of pro-inflammatory mediators and improved mitochondrial function [23]
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However, it's important to balance exercise intensity, as excessive training can temporarily deplete glutathione levels
These effects are not typical but can occur, especially in individuals with pre-existing heart conditions or other risk factors
Verification of ferroptosis and pyroptosis and identification of PTGS2 as the hub gene in human coronary artery atherosclerosis
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