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Mechanistically, GHK-Cu appears to act through several pathways: Collagen and elastin remodeling: Gene array work by Finkley et al., reported in the second edition of Cosmeceuticals and Active Cosmetics (Taylor and Francis, 2005), examined GHK-Cu effects on human fibroblast cultures across a nanomolar to low micromolar concentration range and found upregulation of decorin, collagen, and elastin-associated genes

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They produce greater ROS, protein oxidation, and lipid peroxidation than astrocytes and are more sensitive to unconjugated bilirubin produce death, most likely due to reduced glutathione storage [27]

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