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Under sustained reductions in nutrient inflow, metabolic reserve may narrow as reduced substrate availability limits mitochondrial redox regeneration and constrains the capacity to match oxidative flux with regenerative turnover

Biosimilar pathways may compress prices yet widen access, potentially enlarging patient pools and partially offsetting unit erosion

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7 8 Both drugs have known limitations and side-effect profiles, and neither cures the condition benefits generally require continued use

L-Carnitine plays a critical role in mitochondrial fatty acid transport, enabling -oxidation of long-chain acyl-CoA substrates and efficient ATP generation from lipid stores

When these pathways activate, metabolic rate can shift, and blood vessel responses to cold exposure may change temporarily