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Exenatide treatment ameliorates these morphological alterations and inhibits the mitochondrial apoptotic pathway by suppressing cytochrome c release and favorably modulating the expression of pro-apoptotic (Bax, caspase-3) and anti-apoptotic (Bcl-2) proteins, thereby protecting RGC-5 cells from hyperglycemic injury [43]

Furthermore, Tau has been found to suppress endoplasmic reticulum stress by inhibiting the activation of transcriptional activator 6 and inositol-requiring enzyme-1 molecular pathways (Xu et al., 2017

The warning letters should prompt pharmaceutical companies to reassess their broader ecosystem relationships

Karpova, S

H.MengX.et al (2012)

doi: 10.1007/s11033-014-3252-0