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Experimental and human metabolic literature demonstrates that GH promotes fatty acid mobilization, shifts substrate preference toward fat oxidation, and alters carbohydrate-lipid partitioning during fasting and metabolic stress states, making GH secretagogue systems useful tools for investigating adipose tissue signaling, fuel selection, and the endocrine regulation of energy metabolism [8]

8d) pointing to a critical role for epigenetic modulation in progenitor cell fate 76

There was no significant difference in BMI between the start (43.2 5.9 kg/m 2 ) and stop (41.3 5.2 kg/m 2 ) of GLP-1 therapy

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