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Based on our research, allicin, when used therapeutically, may be delivered into the brain by TRPV2 to improve post-stroke functional impairment, including behavior and the cerebral infarction area

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Ablation of astrocytes, or inhibition of astrocyte specific homoeostatic molecules in experimental animals trigger behavioural abnormalities that resemble neuropsychiatric symptomatology in humans

As estrogen declines, the brain is more sensitive to any additional disruption, including the neurological effects of GLP-1 receptor activation