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Activation of nicotinic acetylcholine receptors can suppress the downstream mechanisms of excitotoxicity, including intracellular Ca 2+ overload, increased ROS, and mitochondrial energy metabolism disorders

Reducing free radical concentrations in tumours allows more vigorous growth

A healthcare provider will decide if this is appropriate based on the patients condition and response to treatment

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Mitochondrial autophagy and cell survival is regulated by the circadian Clock gene in cardiac myocytes during ischemic stress