It relies heavily on glutathione to process toxins and prepare them for removal
For plots c, d, and e, point estimates are represented as circles, and horizontal bars indicate 95% confidence intervals
There is biological plausibility for pancreatic complications due to the supraphysiological stimulation of the pancreas, leading to hyperplasia
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Mitochondrial MDH2 knockdown could have extended mouse lifespan by activating a prolongevity mitochondrial unfolded protein response (UPR mt ) gene expression program that enhances the cellular redox state downstream of initial detrimental redox changes, possibly by inducing the expression of LDH or G3PS enzymes to compensate for the oxidation of the mitochondrial [NAD + ]/[NADH]
