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Therefore, Hurley Stage III is characterized by a self-perpetuating microenvironment that maintains fibrosis, chronic inflammation, and tissue destruction in end-stage HS [77, 89, 91] (Fig

This powerful antioxidant action of GSH led to the conclusion that it is the main mechanism of the cytoprotective action of the N-acetyl-cysteine (NAC), a rather weak antioxidant, acting as GSHs precursor [43,46]

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