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Lipid chain-driven interaction of a lipidated Src-family kinase Lyn with the bilayer membrane

HP = hemoperfusion

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Compared with NRAS activating mutations at positions 12 and 13, NRAS Q61 mutations exert a stronger activating effect on the MAPK pathway since codon 61 is the catalytic residue for GTP hydrolysis and Q61 mutation impedes the return of RAS to an inactive GDP-bound state (92)