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Neutrophil MMP-9 proenzyme, unencumbered by TIMP-1, undergoes efficient activation in vivo and catalytically induces angiogenesis via a basic fibroblast growth factor (FGF-2)/FGFR-2 pathway

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Chou IW, Cheng YH, Chen YR, Hsieh PCH, King K

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[10] Il farmaco in ultima analisi agisce come donatore di gruppi acetilici che, associati con il coenzima A, intervengono nel ciclo di Krebs: vengono cos influenzati sia il metabolismo degli acidi grassi sia quello dei glucidi