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High levels of cytoplasmic tyramine result in its transport into vesicles via VMAT2, displacing stored vesicular norepinephrine into the neuronal cytoplasm, resulting in an increase of cytoplasmic norepinephrine (Raiteri and Levi, 1986

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Although most of the past research has focused on fibrillar A, soluble oligomeric A species are now considered to be of major pathological importance in AD

Treatment effects of intranasal growth hormone releasing peptide-2 in children with short stature

Maintenance treatment led to worsened manic symptoms, especially in patients with a rapid-cycling course [13]

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