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The anti-apoptotic effect was attenuated by PKA inhibition or GLP-1-R knockdown, confirming its dependence on GLP-1-R-PKA/-catenin signaling

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Then, it increased to certain level and reached the saturation point at 4.0 mM of GSH

The retinal damage procedure included L-NAME administration in a total dose of 5 mg/kg, which was given by retrobulbar application in each eye, with 0.5 mg/0.1 ml saline per eye

The levels of these mediators usually exhibit modest correlations, thereby limiting the assignment of molecular meaning and the understanding of global relationships [25]