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The short answer: We dont know
Arbitrary limits invite appeals and frustration
Animal studies have indicated its presence in the area of analysis concerning repair processes, tissue protection and mechanisms related to the integrity of the gastrointestinal tract
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However, the observational nature of the latter studies may hinder biases since T2DM or obesity may be mediating or influential factors acting on the association between GLP-1RAs and AUD and, as a consequence, it is unclear whether the strong effect of these agonists would be still robust in the absence of obesity and/or T2DM