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and Research Grant by the Princess Takamatsu Cancer Research Fund

The peptide exists naturally in human plasma, and supplemental doses remain well below toxic thresholds

Their activity is fine-tuned by GPCR kinases (GRKs), which phosphorylate the receptor and enable -arrestin binding

Given their role as central regulators of vascular homeostasis, EC cuproptosis can initiate widespread dysfunction, impairing barrier integrity and vasoreactivity [153]