Moreover, the nonuniform lung tissue damage leads to the generation of stress concentrations zones and compensatory overexpansion (by remaining alveoli), both of which lead to additional mechanical strain and perpetuate the cycle of damage and progressive airspace enlargement
Shamsad A, Gautam T, Singh R, Banerjee M
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Ung glutathione vi hm lng ln c th gy ng c, vy nn tuyt i khng v mun trng nhanh m s dng vt qu hm lng cho php
[DOI] [PubMed] [Google Scholar] 164.Medeiros R., Baglietto-Vargas D., LaFerla F.M
Many studies have shown that transcription factors such as p53, Nrf2, ATF3, ATF4, YAP1, TAZ, SP1, HIF1A, BACH1, TFEB, JUN and HNF4A play key roles in regulating ferroptosis through transcriptional- or nontranscriptional-dependent mechanisms