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Free radical species produced during oxidative stress are suspected to mediate protein oxidation, lipid peroxidation, and DNA and RNA oxidation in multiple brain regions [150]

Drugs like troglitazone, bromfenac, ticrynafen, benoxaprofen, bromfenac, trovafloxacin, ebrotidine, nimesulide, nefazodone, ximelagatran and pemoline have been withdrawn due to hepatotoxicity [231-233]

Chronic ER stress, resulting from impaired ERAD, triggers the UPR, which, if unresolved, can lead to cardiomyocyte apoptosis and heart failure