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DTCM-g enhanced the radiosensitivity of HOS and MG-63 cells by reducing the regulation of AP-1 on target genes, and inhibiting AP-1 and the expression of PDPN, MMP-2, TIMP1, and TIMP2, which participate in the regulation of extracellular matrix degradation and reconstruction, thereby affecting the occurrence and process of apoptosis 22 (Table 1)