Dopamine-induced oxidative stress, impaired synaptic vesicle function and misfolding of -synuclein, due to mutations or to oxidative damage to this protein, might be components in a self-perpetuating vicious cycle that eventually leads to the demise of dopaminergic neurons
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Concurrently, a decrease in OCR was shown, suggesting a decrease in the activity of the mitochondrial respiratory chain, which is consistent to our results
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