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Trapp and Cork, 2015)

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This elimination of senescent cells occurred selectively, i.e

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It is postulated that viral infection could trigger CD8 T-cell hyperactivation and increased antibody production to compensate for this deficient CD4 T-cell response in genetically predisposed individuals (HLA-DR15), which may lead to autoimmune hypophysitis or direct pituitary damage

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The mechanism of testicular dysfunction is closely related to the above complex pathophysiological changes in diabetes

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From the discovery of the GLP-1 fragment GLP-1(7-37) to the development of more stable and long-acting GLP-1 analogs, these milestones represent significant breakthroughs in the medical field

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