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Potentiation of glucose-induced insulin secretion by GIP is diminished in chronic hyperglycaemic states due at least in part to a decrease in the Gs subtype of the GIP receptor in pancreatic beta cells, and further reasons for concern over GIP as a treatment for type 2 diabetes are that it increases glucagon secretion and adipose deposition [49]

According to the diagnostic criteria described in the Seventh Report of the Joint National Committee of Prevention, Detection, Evaluation and Treatment of High Blood Pressure [24], all of them were hypertensive

19 Thus, patients with T2DM have an impaired responsiveness to GIP with a possible link to GIP-receptor downregulation or desensitization