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Toll-like receptor 4-mediated growth of endometriosis by human heat-shock protein 70

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discovered that downregulation of mTOR in LN patients leads to a reduction in STAT3 and Th17 cells within effector T cells, while inhibiting the AKT signaling pathway hinders the migration of Th17 cells towards inflammatory sites, effectively mitigating the chronic inflammatory process in LN (38, 156)

Simultaneously, GLP-1 may slightly lower resting metabolic rate in some individualsa compensatory mechanism the body employs during weight loss

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