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Our results indicate that VRK2 phosphorylates COPII component Sec24C and promotes its interaction with SLC7A11, thus facilitating ER-to-Golgi transport and membrane expression of SLC7A11

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nutrition, detoxification, de-acidification, antibiotics, bacterial strains and thus the required conditions are almost never obtained

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therefore, downregulation of SIRT1 during aging promotes p53 expression and may increase the vulnerability of neuronal cell deaths