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doi: 10.1371/journal.pone.0151586

pylori infection is coupled to the depletion of intracellular GSH from infected cells

Notably, both diseases share key pathways such as the release of certain inflammatory mediators and oxidative stress, suggesting that therapeutic strategies targeting these common nodes may be beneficial for both conditions, offering a potential for unified treatment approaches

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Furthermore, downregulation of GPX4 via genetic deletion or chemical inhibition can lead to ferroptosis in T cells, while upregulation of GPX4 and genetic deletion of ACSL4 can suppress ferroptosis in CD8+ T cells (88, 89)