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Laboratory studies have also reported changes in growth hormone receptor expression in tendon fibroblasts exposed to BPC-157, suggesting a role in connective-tissue signaling environments [5]

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Although it was speculated that the protective effects of PPAR might depend on its role in facilitating fatty acid catabolism, a recent study showed that this protection is due in part to antioxidant function of the PPAR target gene mitochondrial uncoupling protein 2 (UCP2) [93]

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[DOI] [PubMed] [Google Scholar] 103.Mikolka P., Kopincova J., Mikusiakova L.T., Kosutova P., Calkovska A., Mokra D

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