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Yan M, Huo Y, Yin S, Hu H (2018) Mechanisms of acetaminophen-induced liver injury and its implications for therapeutic interventions

doi:10.3389/fphar.2025.1551843

Cancer cells and inflammatory cells in the tumor microenvironment secrete lymphangiogenic factors, such as VEGF-C (65, 66), VEGF-D (67), PDGF-BB (69), IGF-1 and IGF-2 (75), HGF (76, 77), FGF-2 (78), interleukin-1 (79), and COX-2 (80), promoting the proliferation and dilatation of peritumoral lymphatics, thereby favoring their metastatic spread ( Figure 1 )

A study revealed that cathelicidin-related antimicrobial peptide (CRAMP) derived from PCa facilitates the differentiation of immature myeloid progenitors into macrophages and further polarizes them into the M2 phenotype within the TME (Cha et al., 2016)

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