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Of note, low serum glutamine levels were associated with advanced-stage, higher proinflammatory cytokine levels, poorer overall survival (OS), and progression-free survival (PFS) compared with those with high glutamine levels in colorectal cancer (CRC) patients [70]

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Over time, this can dampen L-cell activity and reduce natural GLP-1 and PYY release

The metabolic byproducts produced by bacteria, such as SCFAs, enhance the release of natural GLP-1, while GLP-1RAs alter signaling pathways, acid responses, immune reactions, and the composition of gut microorganisms, leading to increasingly robust therapeutic feedback mechanisms [108,109]